🔩 Orthopaedic Implants & Principles
Fixation biomechanics, antibiotic prophylaxis, total hip replacement, implant failure, and DVT prophylaxis — core perioperative orthopaedics.
Principles of Fracture Fixation
The fundamental principle of orthopaedic fixation is providing sufficient mechanical stability to allow biological healing. Stability requirements depend on the healing pathway chosen — primary (direct) bone healing requires absolute stability; secondary (indirect) healing via callus requires relative stability. Choosing the wrong fixation type disrupts the intended biology.
🔵 Absolute Stability → Primary Bone Healing
- No movement at fracture site (strain <2%)
- Direct cortical remodelling — Haversian remodelling
- No visible callus on X-ray
- Requires anatomical reduction + rigid fixation
- Methods: Lag screws, compression plating (DCP), absolute rigidity
- Indications: Intra-articular fractures (must be anatomical), simple diaphyseal fractures requiring anatomical reduction
- Lag screw technique: gliding hole in near cortex, thread hole in far cortex → compression across fracture
🟠 Relative Stability → Secondary Bone Healing
- Controlled micro-motion at fracture (strain 2–10%)
- Indirect healing via periosteal callus (endochondral ossification)
- Visible callus on X-ray — reassuring sign of healing
- Biological fixation — preserve soft tissue envelope
- Methods: Intramedullary nails, bridge plating, external fixators, cast immobilisation
- Indications: Comminuted/multi-fragmentary fractures, diaphyseal fractures, open fractures (ex-fix initially)
- Callus = biology working → do NOT disturb
- Load-sharing: Nail + bone share load together. Requires some cortical contact. Used in diaphyseal fractures with cortical contact (e.g., tibial nail, femoral nail). Allows early weight-bearing.
- Load-bearing: Nail bears all load. Used when cortical contact is absent (severely comminuted). Nail takes 100% of load → stress risers at locking screw holes.
- Static locking: Both proximal + distal locking screws — prevents shortening and rotation. Initial locking mode.
- Dynamic locking: Removal of one set of locking screws to allow controlled axial micromotion → stimulates callus. Done if healing delayed (dynamisation).
- Reaming: Widens medullary canal, allows larger nail, improves endosteal blood supply (destroys initially, then promotes via extraosseous collaterals). Reaming may cause fat embolism.
- Common nails: Femoral (antegrade/retrograde), tibial, humeral, PFNA (proximal femoral nail antirotation — for trochanteric NOF fractures)
- Dynamic Compression Plate (DCP): Oval screw holes allow screw eccentrically placed → tightening pulls fracture fragments together (compression). Absolute stability.
- Locking Compression Plate (LCP): Screws lock into plate at fixed angle — like internal fixator. Works without plate-bone contact (preserves periosteum). Relative stability. Used in osteoporotic bone, periarticular fractures.
- Bridge plate: Spans comminuted zone — does not fix intermediate fragments. Relative stability / biological fixation.
- Buttress plate: Resists shear / prevents angular collapse. Used in metaphyseal fractures (distal tibia, proximal tibia).
- Lag screw principle: Near cortex overdrilled (gliding hole) → screw threads only grip far cortex → tightening compresses fracture. Can use cortical screw as lag screw with overdrilling.
- Indications: Open fractures (temporary stabilisation before definitive surgery), damage control orthopaedics (polytrauma), infected nonunion, pelvic ring injuries, soft tissue compromise
- Damage Control Orthopaedics (DCO): Ex-fix in first 24–48 hours → definitive fixation after physiological optimisation (>5 days). Reduces ‘second hit’ phenomenon in polytrauma.
- Complications: Pin site infection, pin loosening, stiffness, delayed conversion to IM nail (increases infection risk if left >2 weeks)
- Ilizarov / circular frame: Fine wire tensioned frames — allows distraction osteogenesis (Ilizarov effect). Used for nonunion, limb lengthening, deformity correction.
- Hemiarthroplasty: One joint surface replaced (e.g., femoral head only in displaced intracapsular NOF fractures in elderly). Faster, less blood loss than THR. Austin-Moore (uncemented) vs Thompson’s (cemented — preferred).
- Total joint replacement: Both surfaces. THR, TKR, TSR.
- Cemented vs cementless: See THR tab.
- Revision arthroplasty: For failed primary replacements. More complex — bone loss, scarring, infection risk.
Biomechanical Principle — Strain Theory (Perren)
Perren’s strain theory: Strain = change in gap / original gap length. Granulation tissue tolerates up to 100% strain. Cartilage: up to 10%. Bone: only 2%. Therefore, a small fracture gap with rigid fixation = very low strain = bone can heal. A large gap with rigid fixation = high strain per unit length = bone cannot bridge. A comminuted fracture with relative fixation distributes strain over many fragments = each sees low strain despite motion. This is why biological bridging fixation works for comminuted fractures.