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Anatomy of the anal canal and sphincter complex; haemorrhoids; anal fistula and Parks' classification; pilonidal sinus; anal fissure; and anal canal tumours.
Layers, muscles, fasciae, rectus sheath and arcuate line, vascular supply and venous drainage, sensory innervation, TAP block anatomy, and abdominal planes.
Anatomy of the vermiform appendix; acute appendicitis — presentation, investigation, and management; appendiceal neoplasms; and the surgical technique of laparoscopic and open appendicectomy.
MRCS oriented biliary anatomy and bile physiology, gallstone pathogenesis, biliary colic, cholecystitis, ascending cholangitis, ERCP, and laparoscopic cholecystectomy technique.
Anatomy of the large intestine; embryological blood supply territories; colonic emergencies — large bowel obstruction, volvulus, stercoral perforation, lower GI haemorrhage, and toxic megacolon.
Which operation for which tumour location — resection extents, vascular ligation targets, surgical planes, anastomotic options, and the rationale behind each oncological procedure.
Inguinal canal — walls, contents, rings, landmarks; spermatic cord contents (Rule of 3); Hesselbach's triangle; hernias compared; femoral triangle and canal; adductor canal.
MRCS oriented Liver anatomy and Couinaud segmentation, clinical assessment, jaundice, portal hypertension and varices, cirrhosis and its complications, and hepatic masses.
Anatomy and physiology, GORD, hiatus hernias, achalasia, Boerhaave's syndrome, Barrett's oesophagus, oesophageal carcinoma, and the three oesophagectomy approaches.
MRCS oriented detailed explanation of pancreatic anatomy, and common pathologies.
MRCS oriented description of anatomy of the duodenum, jejunum and ileum; physiology and absorption; Meckel's diverticulum; small bowel obstruction; Crohn's disease; ischaemia; intestinal fistulas and the SNAP protocol; and tumours of the small bowel.
The spleen is the largest lymphoid organ in the body. It is entirely intraperitoneal and derived embryologically from mesenchymal tissue in the dorsal mesogastrium. In a healthy adult it weighs approximately 75–150 g and is roughly the size of a clenched fist.
Gastric anatomy and physiology, H. pylori, peptic ulcer disease and its complications, gastritis, gastric carcinoma, bariatric surgery, and post-gastrectomy syndromes.
Structure, boundaries, vasculature, lymphatics, nerve supply, Cooper's ligaments, hormonal physiology — MRCS high-yield.
diagnosis, management of carotid artery disease
The spinal cord itself terminates at the conus medullaris, typically at the L1/L2 vertebral level. Below this point, the spinal canal is not empty — it is filled with a bundle of descending nerve roots heading to their respective exit foramina. Because of the mismatch between spinal cord levels and vertebral levels, these roots must travel a long distance downward through the canal before they can exit. Their appearance on imaging — and in the operating theatre — resembles a horse's tail: the cauda equina
The Cerebellopontine Angle, the vestibular schwannoma ("acoustic neuroma"), and how a single expanding tumour knocks out cranial nerves in strict anatomical sequence — CN VIII → CN VII → CN V → posterior fossa
The Circle of Willis at the top, with the vertebral and common carotid arteries supplying it. The right side shows subclavian stenosis causing 'subclavian steal' — blood is retrograde drawn from the vertebral artery.
The ventricular system is a series of interconnected, CSF-filled cavities deep within the brain. CSF flows in a strict, one-way sequence — any obstruction at any point causes the system upstream to balloon with trapped fluid.
MRCS oriented explanation of the central and peripheral controls of breathing
MRCS oriented detailed explanation of the anatomy of cranial nerves and pathologies that can affect them!
Lemierre's syndrome, Ludwig's angina, retropharyngeal abscess, parapharyngeal space infections — the anatomical spread pathways and life-threatening complications.
Cranial nerve exit foramina, triangles of the neck, deep cervical fascia and the danger space — the anatomical foundation for all ENT surgery.
Cranial nerve exit foramina, triangles of the neck, deep cervical fascia and the danger space — the anatomical foundation for all ENT surgery.
MRCS oriented detailed notes on intracranial pressure and management of intracranial hypertension.
Tonsil blood supply, quinsy, tongue innervation, pharyngeal pouch, tracheostomy anatomy, and the tracheo-innominate fistula — the MRCS pharynx and larynx essentials.
Anatomy, embryology, PTH physiology, hyperparathyroidism (primary/secondary/tertiary), hypocalcaemia, surgical management — MRCS high-yield.
Parotid anatomy, facial nerve branches, salivary tumours, Frey's syndrome, submandibular gland excision and calculous disease — the essential MRCS salivary chapter.
Ischaemic vs haemorrhagic stroke, the visual pathway from retina to occipital cortex, and how to localise a lesion from the pattern of visual field loss alone.
Anatomy, embryology, physiology, clinical conditions, thyroid cancers, surgical management and complications — MRCS high-yield.
Course, constrictions, crossings, and the high-yield surgical relations of the ureter that appear repeatedly in the MRCS.
The Brachial Plexus — Roots to Branches: The brachial plexus (C5–T1) supplies the entire upper limb. Trauma injuries split into two syndromes based on the direction of force: downward traction tears upper roots (Erb's), upward traction tears lower roots (Klumpke's). The plexus has five levels: Roots → Trunks → Divisions → Cords → Terminal branches.
Peripheral nerves are vulnerable wherever they run through a tight, non-yielding space — a bony groove, a fibro-osseous tunnel, or a point where they wrap around bone close to the surface. When a fracture occurs at one of these chokepoints, the nerve is stretched, compressed, or severed alongside the bone it hugs.
upper limb vasculature anatomy and common pathologies
common conditions causing hypercoagulability
Vasculitides – MRCS Notes VasculitidesMRCS Part A & B — Comprehensive Study Notes
Urothelial (TCC), squamous cell, adenocarcinoma — risk factors, staging, NMIBC vs MIBC, TURBT, intravesical therapy, radical cystectomy — MRCS high-yield.
Metastases vs primary tumours, gliomas vs meningiomas, clinical presentation, and the neurosurgeon's full toolkit — from dexamethasone to Gamma Knife.
Polyps and premalignant conditions; adenoma-carcinoma sequence; molecular pathways; staging; treatment by stage; advances in systemic therapy; and anal cancer management.
MRCS bank guide to Anatomy of the prostate, urethra , sphincters, BPH, prostate cancer, prostatitis — LUTS, TURP syndrome, PSA, Gleason grading, staging.
Pathology, subtypes, presentation, paraneoplastic syndromes, staging, surgical management, systemic therapy — MRCS high-yield.
Lung cancer histology, TNM staging, surgical resection, RLN anatomy, paraneoplastic syndromes, and the 4 T's of anterior mediastinal masses.
Cardiac output, Frank-Starling law, preload vs afterload, and the Wiggers diagram — the core cardiovascular physiology for MRCS.
Same patient, same spinal cord injury, opposite autonomic crises — one is a complete failure of sympathetic tone, the other is an unregulated explosion of it.
Classify shock by haemodynamic profile — JVP, SVR, skin temperature, and heart rate — and understand the pathophysiology behind each type.
intensive notes on vascular anatomy and physiology
Anatomy, cortex zones, medulla, Cushing's, Conn's, phaeochromocytoma, adrenal insufficiency — MRCS high-yield.
Bone cells, cortical vs cancellous architecture, Wolff's Law, blood supply of long bones, primary vs secondary fracture healing, and bone graft types — fully mapped to MRCS Applied Basic Sciences.
Hypercalcaemia and hypocalcaemia — causes, clinical features, ECG changes, and management — MRCS high-yield.
Ebb & flow phases, hormonal changes, substrate mobilisation, cytokines, and strategies to attenuate the response — Cuthbertson's description — MRCS high-yield.
MEN 1, MEN 2A, MEN 2B — genetics, clinical patterns, surgical protocols, and exam-favourite rules — MRCS high-yield.
Anatomy, embryology, PTH physiology, hyperparathyroidism (primary/secondary/tertiary), hypocalcaemia, surgical management — MRCS high-yield.
SIADH, Diabetes Insipidus (central and nephrogenic), hyponatraemia and hypernatraemia, the water deprivation test — MRCS high-yield.
Understanding pituitary tumour presentations requires knowing the gland's geography intimately. The clinical signs of a growing pituitary mass are entirely dictated by which surrounding structure gets compressed first. The local anatomy is the examination curriculum.
Anatomy of the anal canal and sphincter complex; haemorrhoids; anal fistula and Parks' classification; pilonidal sinus; anal fissure; and anal canal tumours.
Anatomy of the large intestine; embryological blood supply territories; colonic emergencies — large bowel obstruction, volvulus, stercoral perforation, lower GI haemorrhage, and toxic megacolon.
MRCS oriented Liver anatomy and Couinaud segmentation, clinical assessment, jaundice, portal hypertension and varices, cirrhosis and its complications, and hepatic masses.
Anatomy and physiology, GORD, hiatus hernias, achalasia, Boerhaave's syndrome, Barrett's oesophagus, oesophageal carcinoma, and the three oesophagectomy approaches.
Gastric anatomy and physiology, H. pylori, peptic ulcer disease and its complications, gastritis, gastric carcinoma, bariatric surgery, and post-gastrectomy syndromes.
MRCS oriented detailed notes on intracranial pressure and management of intracranial hypertension.
MRCS oriented detailed explanation of skull foramina and structures that exit and enter the skull through them
Weight-based fluid calculations, dehydration assessment, DKA management, and electrolyte disturbances — the essential framework for the MRCS exam and clinical practice.
Urology Basic Sciences . Body fluids, GFR, tubular handling, RAAS, aldosterone, ADH, diuretics, and acid–base — with surgical and clinical context throughout.
pathology, diagnosis and management of renovascular disease, renal artery stenosis
Respiratory centres, central vs peripheral chemoreceptors, the hypoxic drive in COPD, and the danger of high-flow oxygen — core respiratory physiology for MRCS.
MRCS oriented explanation of the central and peripheral controls of breathing
Type I vs Type II respiratory failure, V/Q ratios, right-to-left shunting, dead space, and the alveolar-arterial oxygen gradient — core respiratory physiology for MRCS.
Haemoglobin oxygen carriage, the oxyhaemoglobin dissociation curve, right and left shifts, carbon monoxide poisoning, and 2,3-DPG — the essential O₂ physiology for MRCS.
A urological emergency where hours determine whether the testis survives — and where the clinical examination alone may be sufficient to go straight to theatre.
Key developmental defects of the urinary and reproductive tracts — from penile meatal position to renal fusion. High-yield anatomy, clinical consequences, and management principles for the MRCS.
How children differ from adults anatomically and physiologically — covering the airway, cardiovascular system, respiratory system, fluid balance, renal function, thermoregulation, and age-based normal values relevant to the MRCS examination.
Weight-based fluid calculations, dehydration assessment, DKA management, and electrolyte disturbances — the essential framework for the MRCS exam and clinical practice.
When intracranial pressure takes the path of least resistance — the four herniation types, what gets crushed, and the clinical signs that tell you exactly what is happening.
The UK criteria, preconditions, medico-legal requirements, clinical reflex tests, and the apnoea test — complete and exam-ready.
Developed at the University of Glasgow in 1974 by Teasdale and Jennett, the GCS is the universal language for describing level of consciousness. It gives clinicians a reproducible, objective number that allows serial comparison over time — an 11 that becomes an 8 is an emergency, regardless of who is doing the assessment. Its power lies in its simplicity and speed
MRCS oriented detailed notes on intracranial pressure and management of intracranial hypertension.
Same patient, same spinal cord injury, opposite autonomic crises — one is a complete failure of sympathetic tone, the other is an unregulated explosion of it.
Risk Assessment · ERAS · Bowel Preparation · Surgical Nutrition — MRCS high-yield notes
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When intracranial pressure takes the path of least resistance — the four herniation types, what gets crushed, and the clinical signs that tell you exactly what is happening.
The UK criteria, preconditions, medico-legal requirements, clinical reflex tests, and the apnoea test — complete and exam-ready.
Metastases vs primary tumours, gliomas vs meningiomas, clinical presentation, and the neurosurgeon's full toolkit — from dexamethasone to Gamma Knife.
diagnosis, management of carotid artery disease
The spinal cord itself terminates at the conus medullaris, typically at the L1/L2 vertebral level. Below this point, the spinal canal is not empty — it is filled with a bundle of descending nerve roots heading to their respective exit foramina. Because of the mismatch between spinal cord levels and vertebral levels, these roots must travel a long distance downward through the canal before they can exit. Their appearance on imaging — and in the operating theatre — resembles a horse's tail: the cauda equina
The Cerebellopontine Angle, the vestibular schwannoma ("acoustic neuroma"), and how a single expanding tumour knocks out cranial nerves in strict anatomical sequence — CN VIII → CN VII → CN V → posterior fossa
The Circle of Willis at the top, with the vertebral and common carotid arteries supplying it. The right side shows subclavian stenosis causing 'subclavian steal' — blood is retrograde drawn from the vertebral artery.
The ventricular system is a series of interconnected, CSF-filled cavities deep within the brain. CSF flows in a strict, one-way sequence — any obstruction at any point causes the system upstream to balloon with trapped fluid.
MRCS oriented detailed explanation of the anatomy of cranial nerves and pathologies that can affect them!
Developed at the University of Glasgow in 1974 by Teasdale and Jennett, the GCS is the universal language for describing level of consciousness. It gives clinicians a reproducible, objective number that allows serial comparison over time — an 11 that becomes an 8 is an emergency, regardless of who is doing the assessment. Its power lies in its simplicity and speed
EDH, SDH and SAH — vessels, mechanisms, CT appearances, the four killers of SAH, and everything that will save a life in the trauma bay. Intracranial haemorrhages differ in where the blood collects, what vessel ruptures, and how fast the patient deteriorates. Knowing the space tells you the mechanism; knowing the mechanism tells you the patient and presentation. Three spaces, three completely different emergencies.
MRCS oriented detailed notes on intracranial pressure and management of intracranial hypertension.
The intervertebral disc is like a jam doughnut— a tough outer ring (anulus fibrosus) containing a soft, gelatinous, high-pressure centre (nucleus pulposus). Under degeneration or trauma, the anulus tears and the pressurised nucleus herniates outward. Understanding where it herniates and which nerve it hits is the foundation of the entire topic.
Same patient, same spinal cord injury, opposite autonomic crises — one is a complete failure of sympathetic tone, the other is an unregulated explosion of it.
Understanding pituitary tumour presentations requires knowing the gland's geography intimately. The clinical signs of a growing pituitary mass are entirely dictated by which surrounding structure gets compressed first. The local anatomy is the examination curriculum.
MRCS oriented detailed explanation of skull foramina and structures that exit and enter the skull through them
here are only three tracts you need to understand to derive every spinal cord syndrome from first principles. The key is knowing two things about each: where it travels in the cord (which column), and where it crosses over (decussates). Everything else follows from that.
Ischaemic vs haemorrhagic stroke, the visual pathway from retina to occipital cortex, and how to localise a lesion from the pattern of visual field loss alone.
The fluid-filled central cord cyst that destroys crossing pain fibres from the inside out — producing the classic dissociated sensory loss in a cape distribution.
Anatomy of the anal canal and sphincter complex; haemorrhoids; anal fistula and Parks' classification; pilonidal sinus; anal fissure; and anal canal tumours.
Anatomy of the vermiform appendix; acute appendicitis — presentation, investigation, and management; appendiceal neoplasms; and the surgical technique of laparoscopic and open appendicectomy.
The acute abdomen is defined as abdominal pain of non-traumatic origin lasting less than 5 days. Managing it is less about achieving a precise diagnosis and more about safely identifying and ruling out life-threatening emergencies.
MRCS oriented biliary anatomy and bile physiology, gallstone pathogenesis, biliary colic, cholecystitis, ascending cholangitis, ERCP, and laparoscopic cholecystectomy technique.
Anatomy of the large intestine; embryological blood supply territories; colonic emergencies — large bowel obstruction, volvulus, stercoral perforation, lower GI haemorrhage, and toxic megacolon.
Which operation for which tumour location — resection extents, vascular ligation targets, surgical planes, anastomotic options, and the rationale behind each oncological procedure.
Polyps and premalignant conditions; adenoma-carcinoma sequence; molecular pathways; staging; treatment by stage; advances in systemic therapy; and anal cancer management.
MRCS oriented Liver anatomy and Couinaud segmentation, clinical assessment, jaundice, portal hypertension and varices, cirrhosis and its complications, and hepatic masses.
aetiology, pathology and diagnosis of mesenteric ischaemia
Anatomy and physiology, GORD, hiatus hernias, achalasia, Boerhaave's syndrome, Barrett's oesophagus, oesophageal carcinoma, and the three oesophagectomy approaches.
MRCS oriented detailed explanation of pancreatic anatomy, and common pathologies.
Risk Assessment · ERAS · Bowel Preparation · Surgical Nutrition — MRCS high-yield notes
MRCS oriented description of anatomy of the duodenum, jejunum and ileum; physiology and absorption; Meckel's diverticulum; small bowel obstruction; Crohn's disease; ischaemia; intestinal fistulas and the SNAP protocol; and tumours of the small bowel.
The spleen is the largest lymphoid organ in the body. It is entirely intraperitoneal and derived embryologically from mesenchymal tissue in the dorsal mesogastrium. In a healthy adult it weighs approximately 75–150 g and is roughly the size of a clenched fist.
Gastric anatomy and physiology, H. pylori, peptic ulcer disease and its complications, gastritis, gastric carcinoma, bariatric surgery, and post-gastrectomy syndromes.
The Brachial Plexus — Roots to Branches: The brachial plexus (C5–T1) supplies the entire upper limb. Trauma injuries split into two syndromes based on the direction of force: downward traction tears upper roots (Erb's), upward traction tears lower roots (Klumpke's). The plexus has five levels: Roots → Trunks → Divisions → Cords → Terminal branches.
Compartment syndrome, fat embolism, AVN, CRPS, non-union, and infection — the high-yield complications every surgical trainee must master.
types of amputation and post-op management of amputations
High-yield for MRCS. Know the name, the bone, the key descriptor, and the clinical catch for each.
The intervertebral disc is like a jam doughnut— a tough outer ring (anulus fibrosus) containing a soft, gelatinous, high-pressure centre (nucleus pulposus). Under degeneration or trauma, the anulus tears and the pressurised nucleus herniates outward. Understanding where it herniates and which nerve it hits is the foundation of the entire topic.
Osteoporosis, osteomalacia, hyperparathyroidism, and renal osteodystrophy — biochemistry, radiology, and management for MRCS.
Classification, upper limb nerve deficits, lower limb nerve deficits, the ulnar paradox, and surgical approach nerve risks.
Fixation biomechanics, antibiotic prophylaxis, total hip replacement, implant failure, and DVT prophylaxis — core perioperative orthopaedics.
Peripheral nerves are vulnerable wherever they run through a tight, non-yielding space — a bony groove, a fibro-osseous tunnel, or a point where they wrap around bone close to the surface. When a fracture occurs at one of these chokepoints, the nerve is stretched, compressed, or severed alongside the bone it hugs.
Knee ligaments, meniscal tears, shoulder instability and tendon injuries — the clinical tests are the examiners' favourite.
Anatomy, cortex zones, medulla, Cushing's, Conn's, phaeochromocytoma, adrenal insufficiency — MRCS high-yield.
The Addisonian Crisis in surgical patients — why long-term steroid users need perioperative cover, and how to manage a crisis when it hits.
Breast pain, fibroadenoma, fibrocystic change, breast cysts, duct ectasia, periductal mastitis, intraductal papilloma, fat necrosis, gynaecomastia — MRCS high-yield.
Hypercalcaemia and hypocalcaemia — causes, clinical features, ECG changes, and management. Two metabolic emergencies you must distinguish at a glance.
Foot complications of diabetes
Ebb & flow phases, hormonal changes, substrate mobilisation, cytokines, and strategies to attenuate the response — Cuthbertson's description — MRCS high-yield.
MEN 1, MEN 2A, MEN 2B — genetics, clinical patterns, surgical protocols, and exam-favourite rules — MRCS high-yield.
ADH excess vs ADH absence. Opposite physiology, opposite biochemistry, and a diagnostic test that distinguishes them all.
Anatomy, embryology, physiology, clinical conditions, thyroid cancers, surgical management and complications — MRCS high-yield.
Clinical examination, imaging (mammography + ultrasound), pathological assessment — BIRADS, FNAC, core biopsy, wire-guided biopsy — MRCS high-yield.
Lemierre's syndrome, Ludwig's angina, retropharyngeal abscess, parapharyngeal space infections — the anatomical spread pathways and life-threatening complications.
Cranial nerve exit foramina, triangles of the neck, deep cervical fascia and the danger space — the anatomical foundation for all ENT surgery.
Cranial nerve exit foramina, triangles of the neck, deep cervical fascia and the danger space — the anatomical foundation for all ENT surgery.
Tonsil blood supply, quinsy, tongue innervation, pharyngeal pouch, tracheostomy anatomy, and the tracheo-innominate fistula — the MRCS pharynx and larynx essentials.
Initial assessment, Le Fort fractures, mandibular fractures, orbital blowout, and chemical eye burns — airway first, always.
Anatomy, embryology, PTH physiology, hyperparathyroidism (primary/secondary/tertiary), hypocalcaemia, surgical management — MRCS high-yield.
Parotid anatomy, facial nerve branches, salivary tumours, Frey's syndrome, submandibular gland excision and calculous disease — the essential MRCS salivary chapter.
Anatomy, embryology, physiology, clinical conditions, thyroid cancers, surgical management and complications — MRCS high-yield.
Stanford Type A vs B dissection, DeBakey classification, aortic aneurysm, TEVAR, and the key management principle: IV beta-blockers before vasodilators.
Type I vs Type II respiratory failure, V/Q ratios, right-to-left shunting, dead space, and the alveolar-arterial oxygen gradient — core respiratory physiology for MRCS.
Stable angina, ACS, STEMI — CABG vs PCI indications, the LIMA–LAD graft, and cardiopulmonary bypass principles.
Light's criteria, pleural effusion, empyema staging, pneumothorax management, pleurodesis, and Boerhaave's syndrome — the MRCS pleural and oesophageal essentials.
Lung cancer histology, TNM staging, surgical resection, RLN anatomy, paraneoplastic syndromes, and the 4 T's of anterior mediastinal masses.
A urological emergency where hours determine whether the testis survives — and where the clinical examination alone may be sufficient to go straight to theatre.
Urothelial (TCC), squamous cell, adenocarcinoma — risk factors, staging, NMIBC vs MIBC, TURBT, intravesical therapy, radical cystectomy — MRCS high-yield.
Key developmental defects of the urinary and reproductive tracts — from penile meatal position to renal fusion. High-yield anatomy, clinical consequences, and management principles for the MRCS.
MRCS bank guide to Anatomy of the prostate, urethra , sphincters, BPH, prostate cancer, prostatitis — LUTS, TURP syndrome, PSA, Gleason grading, staging.
Pathology, subtypes, presentation, paraneoplastic syndromes, staging, surgical management, systemic therapy — MRCS high-yield.
pathology, diagnosis and management of renovascular disease, renal artery stenosis
Course, constrictions, crossings, and the high-yield surgical relations of the ureter that appear repeatedly in the MRCS.
incontinence of urine. Five clinical types, two sphincter gates, and the surgical disasters that destroy them — stress, urge, overflow, functional, and the relentless dripping faucet of total incontinence.
Acute urological trauma meets complex pelvic anatomy — renal grading, bladder rupture, the golden rule of urethral injury, and the surgical disasters of penile fracture and testicular rupture. MRCS orientated
acute limb ischaemia aetiology, pathology and management
notes on aetiology, pathology, diagnosis and recognition of aneurysmal disease
notes on aetiology, pathology, diagnosis and management of the vascular emergency of aortic dissection
Stanford Type A vs B dissection, DeBakey classification, aortic aneurysm, TEVAR, and the key management principle: IV beta-blockers before vasodilators.
atherosclerosis pathology, development, and management, and types of pvd and their management.
diagnosis, management of carotid artery disease
high yield MRCS notes summarising commonly used classification systems in vascular surgery
types of amputation and post-op management of amputations
Foot complications of diabetes
aetiology, pathology and diagnosis of mesenteric ischaemia
Rare conditions affecting the arterial systems
upper limb vasculature anatomy and common pathologies
AVF creation and maturation, central venous access, IVC filters, complications, and dialysis catheter management
intensive notes on vascular anatomy and physiology
extensive notes on which investigation to do and when when investigating vascular disease
pharmacology of drugs commonly used in vascular disease
vascular trauma management
Vasculitides – MRCS Notes VasculitidesMRCS Part A & B — Comprehensive Study Notes
common conditions affecting the venous and the lymphatic system
Phases of healing, impaired healing, NPWT, groin wounds, stump care, and advanced wound management for MRCS vascular surgery
abdominal trauma, how to assess it and how to manage it!
EDH, SDH and SAH — vessels, mechanisms, CT appearances, the four killers of SAH, and everything that will save a life in the trauma bay. Intracranial haemorrhages differ in where the blood collects, what vessel ruptures, and how fast the patient deteriorates. Knowing the space tells you the mechanism; knowing the mechanism tells you the patient and presentation. Three spaces, three completely different emergencies.
here are only three tracts you need to understand to derive every spinal cord syndrome from first principles. The key is knowing two things about each: where it travels in the cord (which column), and where it crosses over (decussates). Everything else follows from that.
The spleen is the largest lymphoid organ in the body. It is entirely intraperitoneal and derived embryologically from mesenchymal tissue in the dorsal mesogastrium. In a healthy adult it weighs approximately 75–150 g and is roughly the size of a clenched fist.
Acute urological trauma meets complex pelvic anatomy — renal grading, bladder rupture, the golden rule of urethral injury, and the surgical disasters of penile fracture and testicular rupture. MRCS orientated