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Calcium Homeostasis & Disturbances — MRCS Revision

🧪 Calcium Homeostasis & Disturbances

Hypercalcaemia and hypocalcaemia — causes, clinical features, ECG changes, and management — MRCS high-yield.

Calcium Homeostasis — Overview

Calcium is the most abundant mineral in the body and plays a critical role in neuromuscular function, cardiac rhythm, bone mineralisation, coagulation, and enzyme activation. Serum calcium is tightly regulated between 2.2–2.6 mmol/L by the interplay of PTH, calcitriol (active vitamin D), and calcitonin.

FeatureHypercalcaemia (>2.6 mmol/L)Hypocalcaemia (<2.2 mmol/L)
Most common cause (outpatient)Primary hyperparathyroidismPost-operative (thyroid/parathyroid surgery)
Most common cause (inpatient)MalignancyVitamin D deficiency / chronic kidney disease
Neuromuscular excitability↓ (depressed — muscles and nerves less excitable)↑ (enhanced — muscles and nerves hyperexcitable)
ECGShortened QT intervalProlonged QT interval
Hallmark signs“Stones, Bones, Groans, Psychic Moans”Chvostek’s, Trousseau’s, perioral paraesthesia, stridor
Emergency treatmentIV 0.9% saline first, then bisphosphonatesIV 10% Calcium Gluconate

Corrected Calcium — Always Adjust for Albumin

~40% of serum calcium is bound to albumin — only the ionised (free) fraction is physiologically active. Total serum calcium must be corrected for albumin before interpreting:

Corrected Ca²⁺ = Measured Ca²⁺ + 0.02 × (40 − serum albumin g/L)

Example: Albumin 20 g/L, measured Ca 2.0 mmol/L → corrected Ca = 2.0 + 0.02 × (40−20) = 2.0 + 0.40 = 2.40 mmol/L (normal). Without correction, this patient would be misdiagnosed as hypocalcaemic.

Alternatively, ionised calcium can be measured directly on a blood gas analyser — useful in critically ill patients where albumin is unreliable.

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